Australian Sea Snake envenoming: what you need to know


Australian Sea Snake envenoming: what you need to know

Sea snakes are mainly found in the tropical areas of the Pacific Ocean and the Indian Ocean. They live both near the coast and in the open sea, but prefer the first habitat, which is richer in their prey, fish. Coral formations in particular, where many fish are found, are their favorite habitat.

Unlike fish, sea snakes don't have gills, so they have to periodically surface to breathe. Most species give birth live, near the coast; young are active soon after birth. They have an extremely potent poison, to which fish are particularly sensitive.

Their body demonstrates adaptation to the aquatic environment. The nostrils can be closed hermetically, the head is elongated and wide like the neck to cut through the water, the tail is high and narrow like a fin.

Australian Sea Snake envenoming: what you need to know

Australian Sea Snake Envenoming Causes Myotoxicity and Non-Specific Systemic Symptoms - Australian Snakebite Project, study published on the Frontiers in pharmacology, told us: "Sea snakes are venomous snakes found in the warm parts of the Indo-Pacific, including around Australia.

Most sea snake envenoming causes myotoxicity, but previous Australian case reports describe neurotoxicity. We aimed to describe the epidemiology and clinical presentation of Australian sea snake envenoming and the effectiveness of antivenom.

Patients were recruited to the Australian Snakebite Project (ASP), an Australia-wide prospective observational study recruiting all patients with suspected or confirmed snakebite >2 years.Information about demographics, bite circumstances, species involved, clinical and laboratory features of envenoming, and treatment is collected and entered into a purpose-built database.R Between January 2002 and August 2020, 13 patients with suspected sea snake bite were recruited to ASP, 11 were male; median age was 30 years.

Bites occurred in Queensland and Western Australia.All patients were in or around, coastal waters at the time of bi you. The species involved was identified in two cases. Local effects occurred in 9 patients: pain, swelling, bleeding, bruising.

Envenoming occurred in eight patients and was characterized by non-specific systemic features and myotoxicity. Myotoxicity was severe and rapid in onset in these two patients. Non-specific systemic features included nausea, headache, abdominal pain, and diaphoresis.

Leukocytosis, neutrophilia, and lymphopenia occurred in both patients with myotoxicity and was evident on the first blood test. No patients developed neurotoxicity or coagulopathy. Early Seqirus antivenom therapy was associated with a lower peak creatine kinase.

While relatively rare, sea snake envenoming is associated with significant morbidity and risk of mortality. Early antivenom appears to have a role in preventing severe myotoxicity and should be a goal of therapy."